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General practitioner
Around 40 percent of people suffer from acute or chronic back pain, osteochondrosis, which can last for years. The onset of osteochondrosis is slow and restrained. The first pain occurs during physical activity or sport. The pain can occur anywhere, whether in the back, neck or lower back, and can be accompanied by symptoms such as radiation. At some point, however, the pain remains even at rest and becomes unbearable.
Osteochondrosis is a wear and tear disease of the spine (Scheuermann's disease) and joints. The vertebral bodies and cartilage of the intervertebral discs are affected.
The wear and tear takes place gradually: Constant overloading of the back compresses the intervertebral discs of the spine. This means that at some point the intervertebral discs can no longer regenerate and remain in a compressed state. The surrounding vertebrae are only slightly cushioned, which increases the pressure on individual vertebrae. The vertebra reacts to the constant pressure with cartilage growth at the lateral ends. This reduces the load on the individual vertebra. If the newly formed cartilage surfaces now meet, the typical pain of osteochondrosis occurs. The more these cartilage surfaces grow, the more the mobility of the spine is restricted, which can lead to complete stiffening.
Osteochondrosis manifests itself through several symptoms. Depending on where the cartilage change takes place, the pain occurs in different places.
Basically, however, the following pains arise:
If the spine is affected by osteochondrosis, it is osteochondrosis of the lumbar spine (lws).
This back pain can be caused by incorrect loading of the intervertebral discs. Where exactly this pain occurs depends on which vertebrae were exposed to the incorrect load and how they have changed.
However, sudden acute pain can also be caused by brief periods of incorrect strain. The body compensates for this with a protective posture, which in turn results in tension.
The degree of pain depends on the severity of the incorrect strain and can increase to severe radiation in the arms and legs.
If osteochondrosis occurs in the area of the cervical spine (osteochondrosis hws), the first warning signs may be neck pain and headaches. As the disease progresses, the freedom of movement of the neck may be restricted. In the worst case, torticollis may develop. If the nerves of the spinal canal are also irritated, this can lead to neurological deficits.
In particular, the postures that occur can lead to further tension, which ultimately causes even more pain.
Osteochondrosis results from overloading the intervertebral discs in the spine. The intervertebral disc has a fluid core that absorbs shocks and vibrations between the vertebrae. With increasing age, too little sport and poor nutrition, signs of wear and tear develop. These changes are already visible from the age of twenty. From the age of 60, these changes can be found in the majority of the population. The fluid, shock-absorbing part of the intervertebral disc loses more and more water, which reduces the height of the disc and leaves it in a compressed position.
This reduces the distance between the vertebrae and increases the pressure on them. In order to withstand this increased load, the vertebra reacts with cartilaginous outgrowths on the side - so-called marginal ridges. This allows the increased pressure to be distributed over a larger area. As soon as these marginal ridges touch the other vertebral bodies - during movement - the severe pain of osteochondrosis occurs.
Osteochondrosis is caused by constant poor posture, incorrect loading, heavy physical work and lack of exercise. Osteochondrosis usually only occurs with increasing age as a result of years of incorrect strain. Many occupations are carried out in continuous standing and sitting positions and have the same sequences. As a result, neither the spine nor the back muscles are given the opportunity to relieve themselves or build up protective muscles.
Scoliosis is a disease of the spine in which the spine deviates from the longitudinal axis. This causes the vertebrae to twist. The displaced spine puts one-sided strain on the intervertebral discs and wears them out. The result is that the intervertebral discs lose their protective function for the vertebrae and these are exposed to increased pressure. This in turn creates the opportunity for the cartilaginous spurs of the vertebrae to distribute this pressure. These spurs then cause the pain of osteochondrosis.
Anyone who has a slipped disc at a young age can be affected by osteochondrosis. Even if this herniated disc is treated correctly, it can cause damage that only becomes apparent later. Inflammation of the intervertebral disc also causes the body to adopt a relaxed posture or the vertebrae to shift. The altered position of the vertebrae in turn increases the friction surface, causing the cartilage to lose substance.
In addition, surgery on the intervertebral disc can also increase the incidence of osteochondrosis. However, this happens in very few cases.
Today's diet creates an over-acidified state in the body. Sugar, unhealthy fats and processed foods lead to a base deficit (mineral deficiency/acidosis). The body lacks the necessary minerals, which it extracts from the bones. This leads to a general undersupply of cartilage, bones and joints.
Persistent hyperacidity causes the cartilage to harden and deeper cartilage layers can no longer be adequately supplied with nutrients and degenerate. To prevent this, the body must be supplied with minerals that are as easily utilized as possible by changing the diet. In this way, the alkaline balance can be restored.
Depending on the severity of the osteochondrosis, treatment can initially be carried out without surgical measures.
A sufficient amount of exercise stimulates the lymphatic system and flushes acids and waste products out of the body. In addition, blood circulation throughout the body ensures that the necessary nutrients are transported to the right areas.
Exercise therapy also promotes muscle development, which can strengthen the entire musculoskeletal system and optimize mobility.
The focus here is particularly on strengthening the back and abdominal muscles.
It is particularly important to reduce the inflammation, as otherwise the vertebrae and intervertebral discs will continue to be attacked. This inflammatory reaction should be prevented primarily through diet. Avoiding wheat and sugar is particularly important here. An alkaline diet in combination with the consumption of sufficient omega 3 can also achieve particularly good results.
In order to achieve quick results, targeted support can be provided in the form of nutritional supplements.
Turmeric and omega 3 can be used to reduce the focus of inflammation. You can have your omega 3 content determined with the omega 3 fatty acid analysis.
In order to avoid constant acidosis, it is extremely important to provide the body with the right nutrients through the diet. With an alkaline diet, rapid success can be achieved.
The nutrients green-lipped mussel with glucosamine and chondroitin are recommended for the vertebrae.
Alkaline minerals in the form of powder or capsules are recommended to support rapid deacidification.
Bone support can be achieved with sufficient sunbathing (15-20 minutes without sun cream on the whole body) or vitamin D supplementation. A calcium-rich diet also promotes the positive effects.
Regular relaxation through heat therapies such as red light, mud or massages prove to be particularly helpful. This provides short-term relief for areas that are already under strain.
Sources (in English):
Brayda-Bruno, M., Viganò, M., Cauci, S., Vitale, J. A., de Girolamo, L., De Luca, P. et al (2017, August). Plasma vitamin D and osteo-cartilaginous markers in Italian males affected by intervertebral disc degeneration: Focus on seasonal and pathological trend of type II collagen degradation. Clinica Chimica Acta, 471:87-93. doi: 10.1016/j.cca.2017.05.028
Bruns, J. Werner, M. & Soyka, M. (2016, May). Is vitamin D insufficiency or deficiency related to the development of osteochondritis dissecans? Knee Surgery, Sports Traumatology, Arthroscopy, 24(5):1575-9, doi: 10.1007/s00167-014-3413-7
Liu, N., Chen, Z., Qi, Q. & Shi, Z. (2014, May). The relationship of symptomatic thoracolumbar disc herniation and Scheuermann's disease. European spine journal, 23(5):1059-66, doi: 10.1007/s00586-013-3108-7
Seidler, A., Bolm-Audorff, U., Siol, T., Henkel, N., Fuchs, C., Schug, H. et al. (2003, November). Occupational risk factors for symptomatic lumbar disc herniation; a case-control study. Occupational and Environmental Medicine, 60(11):821-30
Matheson, A. J. & Perry C. M. (2003). Glucosamine: a review of its use in the management of osteoarthritis. Drugs & Aging, 20(14):1041-60
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