Vitamins
Minerals
Amino acids
Antioxidants
Omega-3 fatty acids
Probiotics
Melatonine
Plant substances
Superfoods
Vital mushrooms
Special formulas
Mental health & performance
Fear
Stress, exhaustion & burnout
Mood
Memory & concentration
Immune system
Strenghten the immune system
Allergy
Stomach & intestine
Intestinal cleansing & build-up
Digestive problems
Bloating
Heartburn
Hemorrhoids
Parasites
Bones, joints & muscles
Bones
Joints
Muscles
Beauty
Skin
Hair
Connective tissue
Women's health
Fertility
Hormone balance
PMS
Menopause
Pregnancy & breastfeeding
Libido
Heart health
Heart protection
Blood pressure
Circulation
Cholesterol
Vein health
Sleep & fatigue
Sleep disorder
Jet lag
Energy
Iron deficiency
Liver health
Liver protection
Detoxification
Weight loss & metabolism
Lose weight
Metabolism
Drainage
Men's health
Libido & erectile dysfunction
Prostate
Acid-base balance
Blood sugar
Longevity & cellular regeneration
Eyes & eyesight
Inflammations
Pain
Menge
Subtotal:
Rabatt:
Sie sparen:
Freunde-werben-Freunde Gutschein:
Total:
5 HTP capsules 200mg - from Griffonia - highly dosed - laboratory tested - without undesirable additives
R-alpha lipoic acid - patented R-form - 21x stronger - laboratory tested
Polyneuropathy-Aid-Package - natural treatment - laboratory-tested - effective and sustainable
Artichoke thistle capsules - for effective liver detoxification
Turmeric extract capsules - highly dosed with 95% curcuminoids - equivalent to 10,000mg turmeric - 30x more bioavailable
Intestinal cleansing capsules with psyllium and enzyme complex - for optimal intestinal health
Ginkgo Biloba capsules - 100% pure extract - no inferior powder - laboratory tested & highly dosed
Hair Active Capsules with fenugreek - effective nutrient complex with biotin & zinc - to promote hair growth
Melatonin capsules 5 mg - high-dose - laboratory-tested for fast sleep
MSM capsules - 500mg high-dose methylsulfonylmethane - laboratory tested - 100% ultra pure
Rhodiola capsules -100 % pure extract - high dosage - German premium quality - laboratory tested
Spermidine capsules - wheat germ extract - uniquely HIGH DOSAGE with 8mg spermidine per capsule
Naturopath
NADH is also known as coenzyme 1 and is found in humans, animals and yeast. So basically it is part of every living organism.
Research has confirmed that our NADH levels naturally decline as we age, which can lead to an increase in health problems. These include, for example, the development of chronic diseases, muscle loss and fatigue.
However, some people also use NADH to treat high blood pressure, jet lag, depression, Parkinson's disease or to strengthen the immune system.
➤ Are you curious? Then read on to find out more about NADH.
Alternative anti-ageing products Ansehen
Ansehen
NADH is the abbreviation for nicotinamide adenine dinucleotide. This substance is produced by our body from niacin, a B vitamin.
NADH is known as a "helping molecule" because it binds to other enzymes and triggers reactions in the body that have a positive effect on our health.
According to an article published in Scientific America, "a prominent theory of aging states that mitochondrial deterioration is a key factor in aging."
Mitochondria are specialized structures found in cells. They are involved in many cellular processes and, among other things, help to extract the energy stored in nutrients and convert it into a form of energy that can supply the body's cells with energy.
As mitochondria lose some of their power, this appears to contribute to diseases and symptoms associated with ageing. Examples include heart disease, cognitive decline and fatigue.
However, NADH is involved in energy metabolism and thus also in a number of bodily processes that make life possible. Some research suggests that NADH can restore and improve mitochondrial function. This not only increases energy levels, but also mental clarity, alertness, concentration and memory performance.
➤ Biohackers in particular like to use NADH. They speak of positive experiences in terms of endurance during workouts or running.
There are a number of NADH precursors, such as amino acids and vitamin B3. Precursors are other substances that can be converted into NADH by the body, thereby increasing its levels.
➤However, NADH is not always formed from amino acids.
In contrast to the essential nutrients that we can absorb from food, NADH is produced in the body from freely circulating amino acids. However, our body does not automatically synthesize these into NADH. Instead, the amino acids are broken down and used for various bodily functions.
We need niacin for our body to be able to produce the helpful molecule NADH. The vitamin known as B3 is found in abundance in meat, poultry, oily fish, peanuts, avocados, asparagus, broccoli, potatoes and liver.
This means that only if we supply our body with NADH in the form of food supplements can we be sure that its level will rise.
➤Order vitamin B complex now!
Proponents of NADH supplements believe that they can enhance the natural effects of NADH in the brain.
➤ Some even go so far as to claim that it can restore memory and cognitive function in people with Alzheimer's disease. There is even ongoing research into whether NADH, administered by injection or intravenously, can slow the progression of Parkinson's disease.
We have summarized some of the convincing advantages and their background for you in more detail:
Most of the current research focuses on the use of NADH in the treatment of chronic fatigue syndrome (CFS).
➤ Experts believe that up to one million Americans are affected every year. Women are four times more likely than men to suffer from chronic fatigue syndrome. Women between the ages of 40 and 60 are particularly affected.
A few studies have investigated the combined use of NADH and coenzyme Q10 in people with chronic fatigue syndrome.
➤Buy Ubiquinol capsules now!
NASA investigated the effectiveness of using NADH as a countermeasure for jet lag. The reasoning was that NADH can stimulate dopamine synthesis and thus counteract the effects of jet lag in the form of cognitive dysfunction and drowsiness.
What did studies show?
NASA concluded that "stabilized NADH significantly reduced jetlag-induced cognitive impairment, was easy to administer, and had no adverse side effects."
Anxiety and depression are complex disorders and there are several contributing factors. We know that the chemistry of the brain is altered by stress, anxiety, depression and other illnesses.
Most antidepressant drugs act on the neurotransmitters in the brain. It is assumed that NADH works in a similar but natural way:
Crucially, NADH has a key role in the production of energy in our cells. When NADH levels in our cells are low, nutrients in the brain are depleted and we can experience anxiety, depression, sleep disorders and other mood swings.
Increasing NADH levels has been shown to restore cellular energy, brain chemical levels and the right amount of neuroreceptors and neurotransmitters, bringing the brain back to its normal balance. This influences our mood and regulates the existing imbalance.
➤Order GABA now!
The coenzyme is particularly often associated with Parkinson's disease and touted as a promising treatment.
The theory that NADH could affect Parkinson's disease (PD) was fueled by a 1996 study in which intravenous administration of NADH over eight days led to a temporary improvement in PD symptoms.
Subsequent studies were unable to confirm these results. In some cases, the effects proved to be temporary and were therefore impractical. In others, no noticeable effect on PD symptoms was found. Nevertheless, research into the effect of NADH on Parkinson's is still ongoing.
Experience has shown that NADH is very well tolerated and not harmful to our body, as it produces the substance itself.
However, if used excessively, NADH can cause nervousness, anxiety and insomnia. When administered by injection, NADH can cause pain, swelling and redness at the injection site.
There is little research on the long-term safety of NADH. Although it is considered safe, NADH supplements should not be used in children, pregnant women or breastfeeding mothers.
Matching products Ansehen
Sources (in English):
Alraek, T., Lee, M.S., Choi, TY. et al. Complementary and alternative medicine for patients with chronic fatigue syndrome: A systematic review. BMC Complement Altern Med 11, 87 (2011). doi: 10.1186/1472-6882-11-87
Birkmayer, J. G., Vrecko, C., Volc, D., & Birkmayer, W. (1993). Nicotinamide adenine dinucleotide (NADH)--a new therapeutic approach to Parkinson's disease. Comparison of oral and parenteral application. Acta neurologica Scandinavica. Supplementum, 146, 32–35.
Gong, B., Pan, Y., Vempati, P., Zhao, W., Knable, L., Ho, L., Wang, J., Sastre, M., Ono, K., Sauve, A. A., & Pasinetti, G. M. (2013). Nicotinamide riboside restores cognition through an upregulation of proliferator-activated receptor-γ coactivator 1α regulated β-secretase 1 degradation and mitochondrial gene expression in Alzheimer's mouse models. Neurobiology of aging, 34(6), 1581–1588. doi: 10.1016/j.neurobiolaging.2012.12.005
Bushehri, N., Jarrell, S. T., Lieberman, S., Mirdamadi-Zonozi, N., Birkmayer, G., & Preuss, H. G. (1998). Oral reduced B-nicotinamide adenine dinucleotide (NADH) affects blood pressure, lipid peroxidation, and lipid profile in hypertensive rats (SHR). Geriatric nephrology and urology, 8(2), 95–100. doi: 10.1023/a:1008242900153
Forsyth, L. M., Preuss, H. G., MacDowell, A. L., Chiazze, L., Jr, Birkmayer, G. D., & Bellanti, J. A. (1999). Therapeutic effects of oral NADH on the symptoms of patients with chronic fatigue syndrome. Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology, 82(2), 185–191. doi: 10.1016/S1081-1206(10)62595-1
Kuhn, W., Müller, T., Winkel, R., Danielczik, S., Gerstner, A., Häcker, R., Mattern, C., & Przuntek, H. (1996). Parenteral application of NADH in Parkinson's disease: clinical improvement partially due to stimulation of endogenous levodopa biosynthesis. Journal of neural transmission (Vienna, Austria : 1996), 103(10), 1187–1193. doi: 10.1007/BF01271203
Rainer, M., Kraxberger, E., Haushofer, M., Mucke, H. A., & Jellinger, K. A. (2000). No evidence for cognitive improvement from oral nicotinamide adenine dinucleotide (NADH) in dementia. Journal of neural transmission (Vienna, Austria : 1996), 107(12), 1475–1481. doi: 10.1007/s007020070011
Jesús Castro-Marrero, Mario D. Cordero, María José Segundo, Naia Sáez-Francàs, Natalia Calvo, Lourdes Román-Malo, Luisa Aliste, Tomás Fernández de Sevilla, and José Alegre. Antioxidants & Redox Signaling. Mar 2015.679-685. doi: 10.1089/ars.2014.6181
Swerdlow R. H. (1998). Is NADH effective in the treatment of Parkinson's disease?. Drugs & aging, 13(4), 263–268. doi: 10.2165/00002512-199813040-00002
Choi, W. S., Palmiter, R. D., & Xia, Z. (2011). Loss of mitochondrial complex I activity potentiates dopamine neuron death induced by microtubule dysfunction in a Parkinson's disease model. The Journal of cell biology, 192(5), 873–882. doi: 10.1083/jcb.201009132